Fig. 6

The proposed model for Sb(III) bacterial oxidation in P. steutzeri TS44. In this study, i) the addition of Sb(III) would trigger the ROS-protective system by inducing the transcription of sodB, sodC and katE, with SodB and SodC catalyzing the conversion of ROS to H₂O₂, while KatE responsible for the degradation of excessive H₂O₂ (data not shown); ii) the increased cellular H₂O₂ content enhanced the Sb(III) oxidation rate; and iii) the addition of Sb(III) played a selection role on the characterization of gshA insertion; iv) in addition, the accumulated H₂O₂ is partially cosumed by the upregulated catalase KatE (data not shown)