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Fig. 3 | BMC Microbiology

Fig. 3

From: Target identification in Fusobacterium nucleatum by subtractive genomics approach and enrichment analysis of host-pathogen protein-protein interactions

Fig. 3

The plausible mechanisms in CRC progression due to F. nucleatum infection may involve increased expression of proinflammatory genes, ROS, oncogenes, DNA damaging genes and β-catenin signaling which may contribute to accumulation of mutations that promote tumor induction, proliferation and epithelial-mesenchymal transition

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BMC Microbiology

ISSN: 1471-2180

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