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Fig. 3 | BMC Microbiology

Fig. 3

From: In vivo competition and horizontal gene transfer among distinct Staphylococcus aureus lineages as major drivers for adaptational changes during long-term persistence in humans

Fig. 3

Schemes of the three structural differences on the genomic level between the isolates t012#00 and t021#02 within the mobile elements SaPIn1, SaPIn2, and SA bacteriophage 112C Mu 50 B. Genes on the forward strand are depicted as right-sided arrows; genes located on the reverse strand are depicted as left-sided arrows. Genes showing synteny between both genomes are highlighted with the same color and are linked with a dashed line. Genes that are present in just one of both genomes are colored in red. The locus tags of the genes and the genomic position in relation to the early (for t012#00) and late (for t021#02) genome sequence, respectively, are given and listed in Additional File 7. a Genomic differences within the S. aureus pathogenicity island SaPIn1. In both isolates, the islands are flanked by a gene encoding putative permease (upstream site) and by a gene encoding heat shock protein 60 family chaperone GroEL (downstream site). b Deletion of three genes encoding tandem lipoprotein within pathogenicity islandSaPIn2 in isolate t021#02. c Genomic differences within the shorter copy of phage SA bacteriophage 112C Mu 50 B, which is inserted in the identical genomic region. d Genomic differences within the longer copy of phage SA bacteriophage 112C Mu 50 B, which is inserted in the identical genomic region

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BMC Microbiology

ISSN: 1471-2180

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